Nature Cell Biology

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  • doi:10.1038/s41556-018-0038-y

    Mechanical forces influence both cytoplasmic and nuclear events. Kirby and Lammerding discuss recent evidence suggesting that the nucleus itself is a mechanosensor and methods to study nuclear mechanotransduction.' );return false" > Emerging views of the nucleus as a cellular mechanosensor
  • doi:10.1038/s41556-018-0034-2

    Hawk et al. show that RIPK1 activation during extracellular matrix detachment induces mitophagy through mitochondrial phosphatase PGAM5 to increase reactive oxygen species and non-apoptotic cell death, and that antagonizing RIPK1/PGAM5 enhances tumour formation.' );return false" > RIPK1-mediated induction of mitophagy compromises the viability of extracellular-matrix-detached cells
  • doi:10.1038/s41556-018-0040-4

    Lyden and colleagues use asymmetric flow field-flow fractionation to classify nanoparticles derived from cell lines and human samples, including previously uncharacterized large, Exo-L and small, Exo-S, exosome subsets.' );return false" > Identification of distinct nanoparticles and subsets of extracellular vesicles by asymmetric flow field-flow fractionation
  • doi:10.1038/s41556-018-0055-x

    Publisher Correction: Affinity purification of cell-specific mitochondria from whole animals resolves patterns of genetic mosaicism' );return false" > Publisher Correction: Affinity purification of cell-specific mitochondria from whole animals resolves patterns of genetic mosaicism
  • doi:10.1038/s41556-018-0039-x

    Hervera et al. show that extracellular vesicles containing NOX2 complexes are released from macrophages and incorporated into injured axons, leading to axonal regeneration through PI3K–p-Akt signalling.' );return false" > Reactive oxygen species regulate axonal regeneration through the release of exosomal NADPH oxidase 2 complexes into injured axons
  • doi:10.1038/s41556-018-0044-0

    Author Correction: Sister chromatid resolution is an intrinsic part of chromosome organization in prophase' );return false" > Author Correction: Sister chromatid resolution is an intrinsic part of chromosome organization in prophase
  • doi:10.1038/s41556-017-0033-8

    Xie and colleagues find that activated mTORC1 growth signalling impairs DNA repair through S6K-mediated phosphorylation and inhibition of the RNF168 ligase.' );return false" > The mTOR–S6K pathway links growth signalling to DNA damage response by targeting RNF168
  • doi:10.1038/s41556-017-0030-y

    Using nanopillars with increased spatial resolution, Shiu et al. identify high perinuclear forces that originate from contractile apical actin filaments that span across the nucleus and are dependent on lamin A and the LINC complex.' );return false" > Nanopillar force measurements reveal actin-cap-mediated YAP mechanotransduction
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